Autism is a complex neuro-developmental disorder which is characterised by poor social skills and an obsession with sameness. It roughly affects one in 88 children -- mainly boys -- according to the US Centers for Diseases Control in Atlanta.
The study, which has been funded by the National Institutes of Health in the US, has been published in Cell Press journal Neuron. "These are very exciting results because they suggest that existing drugs might be useful in treatment of the core deficits in autism," said senior author Dr William Catterall of the University of Washington, Seattle.
His team also found that reducing the effectiveness of inhibitory neurons in normal mice also induced some autism-related deficits. "Benzodiazepine drugs had the opposite effect, increasing the activity of inhibitory neurons and diminishing autistic behaviors,'' said a press release put out by the University of Washington.
"Our results provide strong evidence that increasing inhibitory neurotransmission is an effective approach to improvement of social interactions, repetitive behaviors, and cognitive deficits in a well-established animal model of autism," said Dr Catterall.
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